Human ApoE4 haplotype impairs bioenergetic responses to neonatal hypoxia-ischemia and increases neuroinflammation in mice.

Michael Q. Nugent, Johns Hopkins University School of Medicine, Elkridge, MD, United States; Mark St. Pierre, Johns Hopkins University School of Medicine, Waltham, MA, United States; Michelle Nazareth, Johns Hopkins University, Baltimore, MD, United States; Abigail A. Fassinger, Johns Hopkins University, Camillus, NY, United States; Genesis Elmore, Johns Hopkins University School of Medicine, Baltimore, MD, United States; Sarah Ann Duck, Johns Hopkins University School of Medicine, Washington DC, DC, United States; Katherine M. Carlin, United States Air Force, FPO, Armed Forces - AP, United States; Lauren Jantzie, Johns Hopkins University School of Medicine, Baltimore, MD, United States; Lee J. Martin, Johns Hopkins, Baltimore, MD, United States; Frances J.. Northington, Johns Hopkins University School of Medicine, Baltimore, MD, United States; Susan J. Vannucci, susanjvannucci-phd-wellness, New York, NY, United States; Raul Chavez-Valdez, Johns Hopkins University School of Medicine, Baltimore, MD, United States